Author: Darren W.
Source: isnare.com

Acne vulgaris is a skin disorder that most commonly affects sensitive hair follicles, most commonly found in the neck, upper trunk and face. He is described by comedones (which are the primary lesions of acne), which are opened and closed, and cysts, nodules, pustules and papules. Acne is a skin disease more common in adolescents. Both sexes are equally affected, but onset in girls is a little earlier. This may be because girls reach puberty at an earlier age than boys. Acne is most noticeable during adolescence because of the endocrine glands that affect the sebaceous glands (responsible for secretion of sebum or oil) are at their peak performance. Acne seems to be rooted in the interaction of hormonal, bacterial and genetic. The causes of acne vulgaris during childhood, the sebaceous glands are small and almost nonfunctional. These glands are under control of the endocrine system, especially androgens. During puberty, androgens stimulate the sebaceous glands, causing it to enlarge and secrete natural oil called sebum, which rises to the surface of the hair follicle and leaves the skin surface. In adolescents who develop acne, androgenic stimulation produces a heightened response of the sebaceous glands for acne to occur when accumulated sebum plugs pilosebaceuos ducts. This accumulated material forms comedones. The primary lesions of acne are comedones. Closed comedones (whiteheads) are formed from obstructive lesions of lipids or oils and keratin concerned that connect the dilated follicle. They are small, whitish, with openings minutes follicular papules usually can not see. These closed comedones into open comedones may evolve in which the contents of the tubes are in open communication with the external environment. The colored open comedones (blackheads) is not dirt, but an accumulation of lipids, bacteria and epithelial debris. Treatment of Acne Vulgaris Although the exact cause is unknown, some of the whiteheads can break, resulting in an inflammatory reaction caused by leakage of follicular contents (eg, sebum, keratin, bacteria) into the dermis. This inflammatory response can result in the form of the action of certain bacteria such as acnes Proprionibacterium, who lives in the hair follicles and break down the triglycerides of sebum into fatty acids and glycerin. The resulting inflammation seen clinically as erythematous papules, pustules, inflammatory, and inflammatory cysts. Mild papules and cysts drain and heal on their own without treatment. Deeper papules and cysts can result in scarring of the skin. Acne is generally graded as mild, moderate or severe, depending on the number and type of injuries (for example, comedones, papules, pustules, cysts). Acne often leaves small scars on the skin. Acne has different types according to their shape or form. Described as ice pick scars are deep wells, such as volcanoes upside. Box car scars, as its name implies, are the form of tables or angular shapes. Rolling scars appear like waves, while hypertrophic scars or keloid scars are thick. The management objectives are to reduce acne bacteria colonies, decreased activity of the sebaceous glands, hair follicles becoming plugged prevent, reduce inflammation, combating secondary infection, minimize scarring, and eliminate factors that predispose the person to acne. The therapeutic regimen depends on the type of injury. There is no cure for acne vulgaris predictable, but combinations of treatments available that can effectively control its activity.